- Author:Valentin Fuster
- Publisher:Wiley-Blackwell; 1 edition (December 15, 1996)
- Pages:558 pages
- FB2 format1898 kb
- ePUB format1743 kb
- DJVU format1897 kb
- Formats:docx lit lrf mbr
Syndromes of Atherosclerosis book. Syndromes of Atherosclerosis: Correlations of Clinical Imaging and Pathology.
Syndromes of Atherosclerosis book. 087993638X (ISBN13: 9780879936389).
Syndromes of Atherosclerosis: Correlations of Clinical Imaging and Pathology. January 1998 · Chest
Syndromes of Atherosclerosis: Correlations of Clinical Imaging and Pathology. January 1998 · Chest. Interventional cardiology has allowed a correlation between the clinical picture of coronary atherosclerosis with the underlying pathology and has helped establish the evolution of atherosclerotic plaques in vivo. Better understanding of the basic mechanisms of coronary atherosclerosis, due. to the contributions of interventional cardiology, will help cure and/or prevent coronary atherosclerosis in the next few decades. Fuster V, Fallon JT, Badimon JJ, Nemerson Y. The unstable atherosclerotic plaque: clinical significance and therapeutic intervention. Thromb Haemost 1997; 78:247–55. PubMedGoogle Scholar. Armonk, NY: Futura Publishing C. In. 1996:105–22. 65. Feld S, Ganim M, Cavell ES, et al. Comparison of angioscopy intra-vascular ultrasound imaging and quantitative coronary angiography in predicting clinical outcome after coronary intervention in high-risk patients. 87. Small DM, Bond MG, Waugh D, Prack M, Sawyer JK.
Cardio Zone: Dr. Valentin Fuster discusses Heart Rate & BMI at Mount Sinai Health Desk at US Open. Making Rounds: Medical Education Documentary Film.
In: Fuster, V, ed. Atherosclerotic plaque at the carotid artery bifurcation. Correlation of ultrasonographic imaging with morphology. J Ultrasound Med. 1987;6:363–366. 1996:1–15. 1. arins, CK, Giddens, DP, Bharadvaj, BK, Sottiurai, VS, Mabon, RF, Glagov, S. Carotid bifurcation atherosclerosis. Quantitative correlation of plaque localization with flow velocity profiles and wall shear stress. Circ Res. 1983;53:502–514. 'Brien, ER, Schwartz, SM. A new view of restenosis.
1996) in Syndromes of Atherosclerosis: Correlations of Clinical Imaging and Pathology, Angiographic correlations of advanced coronary lesions in acute coronary syndromes, ed Fuster V. (Futura Publishing Company, In. . (Futura Publishing Company, Inc, Armonk, NY), pp 105–122. 2001) New understanding of atherosclerosis (clinically and experimentally) with evolving MRI technology in vivo. Ann N Y Acad Sci 947:181–195, discussion 195–8.
The disease can last for a long time, because treatment often begins with a significant delay. In the absence of therapeutic effects, serious complications may occur, up to tissue necrosis and amputation of the affected limb. Video: Atherosclerosis - Pathophysiology. Description of obliterative disease.
Panel Discussion IIB. Overview of Imaging: Atherosclerotic Regions and Imaging Modalities.
This monograph is a valuable multidisciplinary resource, written by experts in the fields of mechanical and electrical engineering, pathology, neurology, imaging, clinical and vascular biology, physics, radiology and surgery. Panel Discussion I. Overview of Pathology: Clinical Correlations of Atherosclerosis. Panel Discussion IIB.
The Committee on Vascular Lesions also attempted to correlate the appearance of lesions noted in clinical imaging studies with histological lesion types and corresponding clinical syndromes. In the histological classification, lesions are designated by Roman numerals, which indicate the usual sequence of lesions progression. The initial (type I) lesion contains enough atherogenic lipoprotein to elicit an increase in macrophages and formation of scattered macrophage foam cells. As in subsequent lesion types, the changes are more marked in locations of arteries with adaptive intimal thickening.
SummaryRecent clinical, angiographic, and pathologic evidence has prompted reexamination of the natural history of coronary atherosclerosis. Abrupt anatomic complications of atherosclerotic plaques or thrombosis on a plaque may precipitate the malignant syndromes of sudden death, acute myocardial infarction, or unstable angina before the plaque itself is large enough to limit coronary flow. This concept of a bimodal pattern of presentation has important clinical and therapeutic implications.
As a multi-disciplinary resource, this book edited by Valentin Fuster, MD, PhD, chairman of the committee, consists of twenty four chapters written by experts in mechanical and engineering, pathology, neurology, imaging, clinical and vascular biology, physics, radiology and surgery. This invaluable reference tool identifies problems, perspectives and critical new questions for evaluation and dissemination.